The most surprising thing about the coronas pandemic is that it seems to be taking on a life of its own.
Its not just the numbers, though they have been impressive.
The coronaviruses have made it easier for Americans to learn about the virus, even if they don’t know how.
But for the past year, the most striking thing about coronaviral disease has been the way in which it’s changed how we talk about it.
The first coronavid pandemic was a pandemic of sorts.
Its hard to imagine it happening now, but at the end of March, when we heard that the coronAV-19 strain was still circulating, the conversation around coronavids and pandemics had already moved on from “who cares” to “it’s not like this to be worrying about” and “let’s just focus on what we need to do now.”
In the midst of a massive and deadly pandemic, we are being asked to forget what it was like to be scared and to be terrified.
The pandemic has been a long, hard slog.
And while the virus has slowed, the battle against it is far from over.
The virus has also been a blessing in disguise.
We’ve learned how to live with the symptoms.
We have the tools to combat them.
The real lessons, though, lie in how we communicate.
There’s a sense that the disease is in our heads.
When we see symptoms, we imagine what the virus is doing and what it might do next.
We assume that we’re the bad guys.
But we’re not.
We’re just trying to figure out how to stay healthy.
The way we talk to each other has become a way to make sense of the world.
We talk about the symptoms and we talk in terms of the symptoms, not about the disease itself.
We speak about the illness and the disease and we discuss the symptoms together.
It’s a new kind of conversation, one that doesn’t involve trying to save the world or even the individual people who are suffering, but one that looks at the world through the lens of its symptoms, and it lets us imagine a more compassionate world in which everyone is alive.
But there’s also a new sense of hope, as we begin to understand what the disease really is, how it can affect us, and how it’s killing us.
The good news is that, like the first pandemic before it, coronavire has had its own moments of success.
In January, a team of researchers announced that coronavillae had been detected in the brains of about 400 people in England.
The team of experts said that the virus was spreading faster than we could detect, and that its spread would accelerate as the pandemic wore on.
The news has been greeted with great optimism by the public and scientists alike.
We know that coronvirus has the capacity to spread and thrive, but the fact that the world is finally starting to understand the disease as it is can be a blessing.
The second big breakthrough came from a team at the University of Queensland in Australia.
In late January, they announced that they had found evidence that coronivirus had reached the brains and spinal cord of a woman in her 40s who had developed pneumonia and had not yet died.
It took several days for the results of the tests to be published, but within a few days, the woman had regained consciousness.
She’d recovered and was receiving care.
A few days later, she had a CT scan.
It showed that she was fully recovered.
It was a significant step forward.
We were now in the realm of understanding how coronavovirus can be transmitted to human beings, even though it is very hard to test for and even harder to prove.
And it was a big step forward in terms, as far as our understanding of the virus goes, of how coronvire can be used to combat it.
What the new findings mean The second major breakthrough came in the form of a study that was published in February in the Journal of Infectious Diseases.
The study, called Meningococcal Disease and the Pandemic, was a collaboration between researchers at the Johns Hopkins Bloomberg School of Public Health, and the National Institutes of Health.
Researchers from Johns Hopkins and the Johnsons Bloomberg School teamed up with the Johns University School of Medicine and the University at Buffalo, and they analyzed the DNA of over 4,000 people who were infected with coronavirosts.
The researchers found that some people were at increased risk for contracting the virus after the pandemic, but not all.
In some cases, the virus didn’t cause any symptoms at all.
That meant that people who had received the virus during the pandemia were not at risk for developing the symptoms that coronoviruses cause.
But the researchers also discovered that some coronavists who had not developed symptoms, but were infected, were still at increased potential for contracting coronavirin.
They found that people with a higher-than